Proceedings of the International scientific and practical conference ―Science in the Era of Globalization‖ (May 22-24, 2026) / Publisher website: www.naukainfo.com. - Zurich, Switzerland, 2026. - 353 p.

273 prospects. iScience. Elsevier Inc., 2026. Vol. 29, no. 4. Art. 115335. DOI: 10.1016/j.isci.2026.115335 2. Liu S., Xi Q., Li X., Liu H. Mitochondrial dysfunction and alveolar type II epithelial cell senescence: The destroyer and rescuer of idiopathic pulmonary fibrosis. Frontiers in Cell and Developmental Biology. Frontiers Media SA, 2025. Vol. 13. Art. 1535601. DOI: 10.3389/fcell.2025.1535601. 3. Zhao W., Bai B., Li H., Feng Y., Sun J., Fang Y., Zheng P., Zhang G. The role of oxidative stress-related genes in idiopathic pulmonary fibrosis. Scientific Reports. Springer Nature, 2025. Vol. 15, no. 1. Art. 5954. DOI: 10.1038/s41598-025-89770-y. 4. Lan T., Gao H., Chen Y., Liu Y., Wang C., Shi C. RSK1-SRF signaling axis drives fibroblast activation and pulmonary fibrosis: Genetic causality and therapeutic targeting. iScience. Elsevier Inc., 2026. Vol. 29, no. 5. Art. 115495. DOI: 10.1016/j.isci.2026.115495. 5. Yao M., Liu Z., Zhao W., Song S., Huang X., Wang Y. Ferroptosis in idiopathic pulmonary fibrosis: mechanisms, impact, and therapeutic opportunities. Frontiers in Immunology. Frontiers Media SA, 2025. Vol. 16. Art. 1567994. DOI: 10.3389/fimmu.2025.1567994. 6. Qiu X., Bi Q., Wu J., Sun Z., Wang W. Role of ferroptosis in fibrosis: From mechanism to potential therapy. Chinese Medical Journal. Wolters Kluwer Health, 2024. Vol. 137, no. 7. P. 806–817. DOI: 10.1097/CM9.0000000000002784.

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